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You know that little pink thing nestled in the corner of your eye?
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It's actually the remnant of a third eyelid.
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Known as the “plica semilunaris,”
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it's much more prominent in birds and a few mammals,
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and functions like a windshield wiper to keep dust and debris out of their eyes.
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But in humans, it doesn't work.
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It's vestigial, meaning it no longer serves its original purpose.
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There are several other vestigial structures like the plica semilunaris
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in the human body.
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Most of these became vestigial long before homo sapiens existed,
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quietly riding along from one of our ancestor species to the next.
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But why have they stuck around for so long?
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To answer this question, it helps to understand natural selection.
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Natural selection simply means that traits
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which help an organism survive and reproduce in a given environment
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are more likely to make it to the next generation.
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As the environment changes, traits that were once useful can become harmful.
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Those traits are often selected against,
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meaning they gradually disappear from the population.
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But if a trait isn't actively harmful, it might not get selected against,
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and stick around even though it isn't useful.
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Take the tailbone.
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Evolutionary biologists think that as the climate got drier
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and grasslands popped up,
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our tail-bearing ancestors left the trees and started walking on land.
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The tails that had helped them in the trees
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began to disrupt their ability to walk on land.
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So individuals with mutations that reduced the length of their tails
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became more successful at life on land,
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surviving long enough to pass their short tails on to the next generation.
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The change was likely gradual over millions of years until,
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about 20 million years ago,
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our ancestors' external tails disappeared altogether.
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Today, we know human embryos have tails that dissolve as the embryo develops.
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But the stubby tailbone sticks around,
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probably because it doesn't cause any harm—
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in fact, it serves a more minor function
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as the anchor point for certain other muscles.
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Up to 85% of people have a vestigial muscle called the “palmaris longus.”
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To see if you do,
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put your hand down on a flat surface and touch your pinkie to your thumb.
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If you see a little band pop up in the middle of your wrist,
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that's the tendon that attaches to this now-defunct muscle.
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In this case, the fact that not everyone has it has helped us trace its function.
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Vestigial traits can persist when there's no incentive to lose them—
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but since there's also no incentive to keep them,
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random mutations will sometimes still eliminate them
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from part of the population.
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Looking at our primate relatives,
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we can see that the palmaris longus is sometimes absent
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in those that spend more time on the land,
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but always present in those that spend more time in trees.
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So we think it used to help us swing from branch to branch,
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and became unnecessary when we moved down to land.
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The appendix, meanwhile, may once have been part of the intestinal system
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our ancestors used for digesting plant materials.
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As their diets changed, those parts of the intestinal system began to shrink.
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Unlike other vestigial structures, though, the appendix isn't always harmless—
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it can become dangerously inflamed.
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For most of human history, a burst appendix could be a death sentence.
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So why did it stick around?
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It's possible that it was very slowly on its way out,
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or that mutations simply hadn't arisen to make it smaller.
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Or maybe it has other benefits—
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for example, it might still be a reservoir of bacteria that helps us break down food.
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But the fact is, we're not really sure why the appendix persists.
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Evolution is an imperfect process.
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Human beings are the result of millions of years of trial, error, and random chance—
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and we're full of evolutionary relics to remind us of that.